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Lipid metabolism. It activates LDL receptors in the liver and changes the profile of lipids and lipoproteins in the blood, leading to a decrease in the blood of LDL, apolipoprotein B, and the concentration of Xc.
Metabolism of carbohydrates. Increases insulin release, but fasting glucose concentration usually does not change. In children with hypopituitarism, fasting hypoglycemia may develop. This condition is reversible with the introduction of somatropin.
Water and mineral exchange. Growth hormone deficiency is associated with decreased plasma and tissue fluid volume. Both of these indicators increase rapidly after treatment with somatropin.
Bone metabolism. Stimulates bone metabolism. In patients with growth hormone deficiency and osteoporosis, long-term therapy with somatropin leads to the normalization of the mineral composition and bone density.
Physical performance. Somatropin treatment increases muscle strength and physical endurance. Cardiac output also increases, but the mechanism of this effect has not yet been clarified. A decrease in TPRV can play a certain role in this.
Mental condition. In patients with growth hormone deficiency, there may be a decrease in mental capacity and a change in mental status. Somatropin increases vitality, improves memory and affects the balance of neurotransmitters in the brain.